What kind of evidence is included on the Semax safety evidence page?

Only PubMed-verified publications and explicitly labeled editorial-review gaps are included.

Does this page provide medical advice or human-use guidance?

No. Atlas provides an educational publication map and does not provide sourcing, human-use instructions, or individual medical guidance.

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Semax: Safety evidence

What the verified publication set can and cannot establish about safety.

Safety evidence boundary

Atlas does not infer safety from absence of an adverse-event statement. The records below are included only when the verified publication set provides relevant human context or identifies material design limitations.

Publication records

15 verified studies

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Grade BHuman observational study2018Indexed abstract extracted

The efficacy of Semax in the treatment of patients at different stages of ischemic stroke

A rehabilitation-stage comparison that followed 110 people after ischemic stroke and assessed plasma BDNF, motor performance, and Barthel index scores.

Journal: Zhurnal nevrologii i psikhiatrii imeni S.S. Korsakova
Evidence context: Human comparative or observational evidence

Safety evidence boundary

15 verified studies

The efficacy of Semax in the treatment of patients at different stages of ischemic stroke

Effects of Semax on the Default Mode Network of the Brain

Brain Protein Expression Profile Confirms the Protective Effect of the ACTH(4-7)PGP Peptide (Semax) in a Rat Model of Cerebral Ischemia-Reperfusion

Novel Insights into the Protective Properties of ACTH(4-7)PGP (Semax) Peptide at the Transcriptome Level Following Cerebral Ischaemia-Reperfusion in Rats

Semax, a Synthetic Regulatory Peptide, Affects Copper-Induced Abeta Aggregation and Amyloid Formation in Artificial Membrane Models

Semax peptide targets the μ opioid receptor gene Oprm1 to promote deubiquitination and functional recovery after spinal cord injury in female mice.

Semax, synthetic ACTH(4-10) analogue, attenuates behavioural and neurochemical alterations following early-life fluvoxamine exposure in white rats.

Semax, an ACTH(4-10) analogue with nootropic properties, activates dopaminergic and serotoninergic brain systems in rodents.

Semax and Pro-Gly-Pro activate the transcription of neurotrophins and their receptor genes after cerebral ischemia.

Semax, a Copper Chelator Peptide, Decreases the Cu(II)-Catalyzed ROS Production and Cytotoxicity of aβ by Metal Ion Stripping and Redox Silencing.

Semax, an analogue of adrenocorticotropin (4-10), binds specifically and increases levels of brain-derived neurotrophic factor protein in rat basal forebrain.

Semax, an analog of ACTH((4-7)), regulates expression of immune response genes during ischemic brain injury in rats.

Semax prevents learning and memory inhibition by heavy metals.

Functional Connectomic Approach to Studying Selank and Semax Effects.

The peptide semax affects the expression of genes related to the immune and vascular systems in rat brain focal ischemia: genome-wide transcriptional analysis.

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