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Atlas Research Compounds

Semax: Research history

A publication timeline built only from verified study records.

Verified publication timeline

  1. Semax, an ACTH(4-10) analogue with nootropic properties, activates dopaminergic and serotoninergic brain systems in rodents.Study →
  2. Semax, an analogue of adrenocorticotropin (4-10), binds specifically and increases levels of brain-derived neurotrophic factor protein in rat basal forebrain.Study →
  3. Semax and Pro-Gly-Pro activate the transcription of neurotrophins and their receptor genes after cerebral ischemia.Study →
  4. The peptide semax affects the expression of genes related to the immune and vascular systems in rat brain focal ischemia: genome-wide transcriptional analysis.Study →
  5. Semax prevents learning and memory inhibition by heavy metals.Study →
  6. Semax, an analog of ACTH((4-7)), regulates expression of immune response genes during ischemic brain injury in rats.Study →
  7. The efficacy of Semax in the treatment of patients at different stages of ischemic strokeStudy →
  8. Effects of Semax on the Default Mode Network of the BrainStudy →
  9. Novel Insights into the Protective Properties of ACTH(4-7)PGP (Semax) Peptide at the Transcriptome Level Following Cerebral Ischaemia-Reperfusion in RatsStudy →
  10. Functional Connectomic Approach to Studying Selank and Semax Effects.Study →
  11. Brain Protein Expression Profile Confirms the Protective Effect of the ACTH(4-7)PGP Peptide (Semax) in a Rat Model of Cerebral Ischemia-ReperfusionStudy →
  12. Semax, synthetic ACTH(4-10) analogue, attenuates behavioural and neurochemical alterations following early-life fluvoxamine exposure in white rats.Study →
  13. Semax, a Synthetic Regulatory Peptide, Affects Copper-Induced Abeta Aggregation and Amyloid Formation in Artificial Membrane ModelsStudy →
  14. Semax peptide targets the μ opioid receptor gene Oprm1 to promote deubiquitination and functional recovery after spinal cord injury in female mice.Study →
  15. Semax, a Copper Chelator Peptide, Decreases the Cu(II)-Catalyzed ROS Production and Cytotoxicity of aβ by Metal Ion Stripping and Redox Silencing.Study →

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